Mechanisms of cytoplasmic {beta}-catenin accumulation and its involvement in tumorigenic activities mediated by oncogenic splicing variant of the receptor originated from Nantes tyrosine kinase. [electronic resource]
Producer: 20050816Description: 25087-94 p. digitalISSN:- 0021-9258
- Active Transport, Cell Nucleus
- Alternative Splicing
- Androstadienes -- pharmacology
- Animals
- Blotting, Western
- Cell Nucleus -- metabolism
- Cyclin D1 -- metabolism
- Cytoplasm -- metabolism
- Cytoskeletal Proteins -- biosynthesis
- Enzyme Inhibitors -- pharmacology
- Gene Silencing
- Glycogen Synthase Kinase 3 -- metabolism
- Glycogen Synthase Kinase 3 beta
- Mice
- Models, Biological
- NIH 3T3 Cells
- Neoplasms -- metabolism
- Phosphorylation
- Protein-Tyrosine Kinases -- chemistry
- Proto-Oncogene Proteins c-myc -- metabolism
- RNA, Messenger -- metabolism
- RNA, Small Interfering -- metabolism
- Receptor Protein-Tyrosine Kinases -- chemistry
- Serine -- chemistry
- Trans-Activators -- biosynthesis
- Tyrosine -- chemistry
- Wortmannin
- beta Catenin
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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