Functional genomics identifies a mechanism for estrogen activation of the retinoic acid receptor alpha1 gene in breast cancer cells. [electronic resource]
Producer: 20050914Description: 1584-92 p. digitalISSN:- 0888-8809
- Acetyltransferases -- metabolism
- Base Sequence
- Breast Neoplasms -- metabolism
- Cell Line, Tumor
- Chromatin Immunoprecipitation
- Cloning, Molecular
- Conserved Sequence
- Estradiol -- metabolism
- Estrogen Receptor alpha -- metabolism
- Estrogens -- metabolism
- Genome
- Histone Acetyltransferases
- Humans
- Introns
- Molecular Sequence Data
- Nuclear Receptor Coactivator 3
- Nuclear Receptor Subfamily 1, Group F, Member 1
- Oncogene Proteins -- metabolism
- Plasmids -- metabolism
- Promoter Regions, Genetic
- Protein Binding
- Receptor Protein-Tyrosine Kinases
- Receptor Tyrosine Kinase-like Orphan Receptors
- Receptors, Cell Surface -- genetics
- Receptors, Cytoplasmic and Nuclear
- Response Elements
- Reverse Transcriptase Polymerase Chain Reaction
- Sp1 Transcription Factor -- metabolism
- Trans-Activators -- metabolism
- Transcription, Genetic
- Transfection
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
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