Astrocytes regulate inhibitory synapse formation via Trk-mediated modulation of postsynaptic GABAA receptors. [electronic resource]
Producer: 20060301Description: 3638-50 p. digitalISSN:- 1529-2401
- Analysis of Variance
- Animals
- Animals, Newborn
- Astrocytes -- cytology
- Biotinylation -- methods
- Blotting, Western -- methods
- Brain-Derived Neurotrophic Factor -- physiology
- Cell Count -- methods
- Cells, Cultured
- Coculture Techniques -- methods
- Culture Media, Conditioned -- pharmacology
- Electric Stimulation -- methods
- Embryo, Mammalian
- Excitatory Postsynaptic Potentials -- drug effects
- Gene Expression Regulation -- drug effects
- Glial Fibrillary Acidic Protein -- metabolism
- Hippocampus -- cytology
- Immunoglobulin G -- pharmacology
- Immunohistochemistry -- methods
- In Situ Nick-End Labeling -- methods
- Mice
- Mice, Knockout
- Microtubule-Associated Proteins -- metabolism
- Nerve Growth Factors -- pharmacology
- Neural Inhibition -- drug effects
- Neurons -- cytology
- Patch-Clamp Techniques -- methods
- Presynaptic Terminals -- drug effects
- Rats
- Rats, Sprague-Dawley
- Receptor, trkA -- deficiency
- Receptors, GABA-A -- physiology
- Synapses -- drug effects
- Synaptophysin -- metabolism
- Time Factors
- Vesicular Glutamate Transport Protein 1 -- metabolism
- Vesicular Inhibitory Amino Acid Transport Proteins -- metabolism
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Publication Type: Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
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