Mycobacterium tuberculosis heat shock proteins use diverse Toll-like receptor pathways to activate pro-inflammatory signals. [electronic resource]
Producer: 20050712Description: 20961-7 p. digitalISSN:- 0021-9258
- Animals
- Bacterial Proteins -- chemistry
- Bone Marrow Cells -- cytology
- Cell Line
- Cells, Cultured
- Chaperonin 60
- Chaperonins -- chemistry
- DNA, Complementary -- metabolism
- Dose-Response Relationship, Drug
- Endothelium, Vascular -- metabolism
- Genes, Dominant
- HSP70 Heat-Shock Proteins -- chemistry
- Heat-Shock Proteins -- metabolism
- Humans
- Immune System
- Inflammation
- Lipopolysaccharide Receptors -- chemistry
- Macrophages -- cytology
- Membrane Glycoproteins -- metabolism
- Mice
- Mycobacterium tuberculosis -- metabolism
- NF-kappa B -- metabolism
- Peptides -- chemistry
- Phagocytosis
- Receptors, Cell Surface -- metabolism
- Recombinant Proteins -- chemistry
- Skin -- metabolism
- Toll-Like Receptor 2
- Toll-Like Receptor 4
- Toll-Like Receptors
- Transfection
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, P.H.S.
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