Human endothelial nitric oxide synthase gene delivery protects against cardiac remodeling and reduces oxidative stress after myocardial infarction. [electronic resource]
Producer: 20050418Description: 2457-71 p. digitalISSN:- 0024-3205
- Adenoviridae
- Animals
- Apoptosis -- physiology
- Azo Compounds
- Caspase 3
- Caspases -- metabolism
- Cell Cycle Proteins -- metabolism
- Collagen
- Coronary Vessels -- surgery
- Cyclic GMP -- metabolism
- Cyclin-Dependent Kinase Inhibitor p27
- Genetic Therapy -- methods
- Genetic Vectors
- Heart Ventricles -- pathology
- Humans
- Immunoblotting
- Immunohistochemistry
- In Situ Nick-End Labeling
- JNK Mitogen-Activated Protein Kinases -- metabolism
- Ligation
- Luciferases -- genetics
- MAP Kinase Kinase 4
- Male
- Mitogen-Activated Protein Kinase Kinases -- metabolism
- Myocardial Infarction -- therapy
- NAD -- metabolism
- NADP -- metabolism
- NF-kappa B -- metabolism
- Nitric Oxide -- metabolism
- Nitric Oxide Synthase -- genetics
- Nitric Oxide Synthase Type III
- Organ Size
- Oxidative Stress -- genetics
- Rats
- Rats, Wistar
- Superoxides -- metabolism
- Transforming Growth Factor beta -- metabolism
- Transforming Growth Factor beta1
- Tumor Suppressor Proteins -- metabolism
- Ventricular Function
- Ventricular Remodeling -- genetics
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Publication Type: Comparative Study; Journal Article; Research Support, U.S. Gov't, P.H.S.
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