Toll-like receptor 4 on nonhematopoietic cells sustains CNS inflammation during endotoxemia, independent of systemic cytokines. [electronic resource]
Producer: 20051013Description: 1788-96 p. digitalISSN:- 1529-2401
- Animals
- Bone Marrow Transplantation
- Brain -- metabolism
- CX3C Chemokine Receptor 1
- Cell Lineage
- Choroid Plexus -- metabolism
- Corticosterone -- blood
- Cytokines -- physiology
- Encephalitis -- etiology
- Endotoxemia -- complications
- Gene Expression Regulation
- Hypothalamo-Hypophyseal System -- physiopathology
- I-kappa B Proteins -- biosynthesis
- Lipopolysaccharides -- toxicity
- Male
- Meninges -- metabolism
- Mice
- Mice, Inbred C3H
- Mice, Inbred C57BL
- NF-KappaB Inhibitor alpha
- Pituitary-Adrenal System -- physiopathology
- RNA, Messenger -- biosynthesis
- Radiation Chimera
- Receptors, Chemokine -- biosynthesis
- Receptors, Immunologic -- biosynthesis
- Spleen -- metabolism
- Toll-Like Receptor 4
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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