Constitutive activation of phosphatidyl-inositide 3 kinase contributes to the survival of Hodgkin's lymphoma cells through a mechanism involving Akt kinase and mTOR. [electronic resource]
Producer: 20050421Description: 498-506 p. digitalISSN:- 0022-3417
- Adaptor Proteins, Signal Transducing
- Antibiotics, Antineoplastic -- pharmacology
- Carrier Proteins -- metabolism
- Cell Cycle Proteins
- Cell Line, Tumor
- Cell Survival -- drug effects
- Chromones -- pharmacology
- Enzyme Inhibitors -- pharmacology
- Glycogen Synthase Kinase 3 -- metabolism
- Hodgkin Disease -- enzymology
- Humans
- Immunohistochemistry -- methods
- Morpholines -- pharmacology
- Phosphatidylinositol 3-Kinases -- metabolism
- Phosphoproteins -- metabolism
- Phosphorylation
- Protein Kinases -- metabolism
- Protein Serine-Threonine Kinases -- metabolism
- Proto-Oncogene Proteins -- metabolism
- Proto-Oncogene Proteins c-akt
- Ribosomal Protein S6 Kinases, 70-kDa -- metabolism
- Sirolimus -- pharmacology
- TOR Serine-Threonine Kinases
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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