Mineralocorticoid receptor inhibition ameliorates the transition to myocardial failure and decreases oxidative stress and inflammation in mice with chronic pressure overload. [electronic resource]
Producer: 20050711Description: 420-7 p. digitalISSN:- 1524-4539
- Animals
- Aorta
- Apoptosis
- Blood Pressure
- Cell Size
- Chronic Disease
- Constriction, Pathologic -- complications
- Drug Evaluation, Preclinical
- Eplerenone
- Fibrosis
- Heart Failure -- etiology
- Hypertrophy, Left Ventricular -- complications
- Intercellular Adhesion Molecule-1 -- analysis
- Ligation
- Male
- Matrix Metalloproteinases -- analysis
- Mice
- Mineralocorticoid Receptor Antagonists
- Myocarditis -- drug therapy
- Myocardium -- chemistry
- Myocytes, Cardiac -- pathology
- Oxidative Stress -- drug effects
- Pressure
- Random Allocation
- Receptors, Mineralocorticoid -- physiology
- Spironolactone -- analogs & derivatives
- Tissue Inhibitor of Metalloproteinase-1 -- analysis
- Tissue Inhibitor of Metalloproteinase-2 -- analysis
- Tyrosine -- analogs & derivatives
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
There are no comments on this title.
Log in to your account to post a comment.