Diminished Akt phosphorylation in neurons lacking glutathione peroxidase-1 (Gpx1) leads to increased susceptibility to oxidative stress-induced cell death. [electronic resource]
Producer: 20050526Description: 283-93 p. digitalISSN:- 0022-3042
- Animals
- Apoptosis -- drug effects
- Brain Ischemia -- genetics
- Carrier Proteins -- metabolism
- Cell Death -- drug effects
- Cells, Cultured
- Enzyme Activation -- drug effects
- Genetic Predisposition to Disease
- Glutathione Peroxidase -- genetics
- Hydrogen Peroxide -- toxicity
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Nerve Growth Factor -- pharmacology
- Neurons -- drug effects
- Oxidants -- toxicity
- Oxidative Stress -- physiology
- Phosphatidylinositol 3-Kinases -- metabolism
- Phosphorylation
- Protein Serine-Threonine Kinases -- metabolism
- Proto-Oncogene Proteins -- metabolism
- Proto-Oncogene Proteins c-akt
- bcl-Associated Death Protein
- Glutathione Peroxidase GPX1
No physical items for this record
Publication Type: Journal Article
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