Postconditioning inhibits mitochondrial permeability transition. [electronic resource]
Producer: 20050712Description: 194-7 p. digitalISSN:- 1524-4539
- Animals
- Apoptosis -- drug effects
- Calcium -- metabolism
- Coronary Disease -- physiopathology
- Cyclosporine -- therapeutic use
- Extracellular Signal-Regulated MAP Kinases -- physiology
- Ion Channel Gating -- drug effects
- Ion Channels -- drug effects
- Ion Transport -- drug effects
- MAP Kinase Signaling System -- drug effects
- Male
- Membrane Potentials
- Mitochondria, Heart -- drug effects
- Mitochondrial Membrane Transport Proteins
- Mitochondrial Permeability Transition Pore
- Myocardial Infarction -- etiology
- Myocardial Ischemia -- complications
- Myocardial Reperfusion Injury -- enzymology
- Necrosis
- Oxidative Stress
- Protein Serine-Threonine Kinases -- physiology
- Proto-Oncogene Proteins -- physiology
- Proto-Oncogene Proteins c-akt
- Rabbits
- Random Allocation
- Time Factors
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Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
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