Pharmacological inhibition and genetic deficiency of plasminogen activator inhibitor-1 attenuates angiotensin II/salt-induced aortic remodeling. [electronic resource]
Producer: 20050802Description: 365-71 p. digitalISSN:- 1524-4636
- Acetates -- pharmacology
- Administration, Oral
- Angiotensin II -- toxicity
- Animals
- Antigens, Differentiation -- biosynthesis
- Aorta -- drug effects
- Aortic Diseases -- chemically induced
- Blood Pressure -- drug effects
- Chemokine CCL2 -- biosynthesis
- Collagen Type I -- biosynthesis
- Collagen Type III -- biosynthesis
- Drug Evaluation, Preclinical
- Fibronectins -- biosynthesis
- Fibrosis
- Gene Expression Regulation -- drug effects
- Glomerulosclerosis, Focal Segmental -- chemically induced
- Heart -- drug effects
- Hypertrophy, Left Ventricular -- chemically induced
- Indoleacetic Acids
- Indoles -- pharmacology
- Kidney -- drug effects
- Male
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Myocardium -- metabolism
- Nephrectomy
- Osteopontin
- Plasminogen Activator Inhibitor 1 -- deficiency
- RNA, Messenger -- biosynthesis
- Random Allocation
- Sialoglycoproteins -- biosynthesis
- Single-Blind Method
- Sodium Chloride, Dietary -- toxicity
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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