Phosphatidylinositol 3-kinase offsets cAMP-mediated positive inotropic effect via inhibiting Ca2+ influx in cardiomyocytes. [electronic resource]
Producer: 20050706Description: 1183-90 p. digitalISSN:- 1524-4571
- Androstadienes -- pharmacology
- Animals
- Calcium -- metabolism
- Calcium Channels, L-Type -- metabolism
- Calcium Signaling -- physiology
- Cells, Cultured -- drug effects
- Chromones -- pharmacology
- Colforsin -- pharmacology
- Cyclic AMP -- antagonists & inhibitors
- Cyclic AMP-Dependent Protein Kinases -- pharmacology
- Enzyme Activation -- drug effects
- GTP-Binding Protein alpha Subunits, Gi-Go -- antagonists & inhibitors
- GTP-Binding Protein beta Subunits -- physiology
- GTP-Binding Protein gamma Subunits -- physiology
- Insulin-Like Growth Factor I -- pharmacology
- Ion Transport -- drug effects
- Isoproterenol -- pharmacology
- Morpholines -- pharmacology
- Myocardial Contraction
- Myocytes, Cardiac -- drug effects
- Norepinephrine -- pharmacology
- Patch-Clamp Techniques
- Peptide Fragments -- pharmacology
- Pertussis Toxin -- pharmacology
- Phosphatidylinositol 3-Kinases -- physiology
- Phosphoinositide-3 Kinase Inhibitors
- Phosphorylation -- drug effects
- Prazosin -- pharmacology
- Propanolamines -- pharmacology
- Protein Processing, Post-Translational -- drug effects
- Rats
- Receptors, Adrenergic, beta-1 -- physiology
- Recombinant Proteins -- pharmacology
- Second Messenger Systems -- physiology
- Signal Transduction -- physiology
- Wortmannin
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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