Crucial role of the CCL2/CCR2 axis in neointimal hyperplasia after arterial injury in hyperlipidemic mice involves early monocyte recruitment and CCL2 presentation on platelets. [electronic resource]
Producer: 20050615Description: 1125-33 p. digitalISSN:- 1524-4571
- Animals
- Antibodies, Monoclonal -- pharmacology
- Apolipoproteins E -- deficiency
- Blood Platelets -- chemistry
- Carotid Artery Injuries -- blood
- Carotid Artery, Common -- chemistry
- Cell Adhesion -- physiology
- Cell Movement
- Cells, Cultured -- drug effects
- Chemokine CCL2 -- analysis
- Chemokines -- blood
- Female
- Hyperlipoproteinemia Type II -- genetics
- Hyperplasia
- Macrophages -- pathology
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Monocytes -- pathology
- Muscle, Smooth, Vascular -- cytology
- Myocytes, Smooth Muscle -- chemistry
- Platelet Adhesiveness
- Protein Binding
- Receptors, CCR2
- Receptors, Chemokine -- deficiency
- Recombinant Proteins -- blood
- Time Factors
- Tumor Necrosis Factor-alpha -- pharmacology
- Tunica Intima -- pathology
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.