Nogo-66 receptor prevents raphespinal and rubrospinal axon regeneration and limits functional recovery from spinal cord injury. [electronic resource]
Producer: 20050118Description: 439-51 p. digitalISSN:- 0896-6273
- 5,7-Dihydroxytryptamine -- pharmacology
- Animals
- Axons -- physiology
- Behavior, Animal
- Blotting, Northern -- methods
- Blotting, Southern -- methods
- Brain -- pathology
- Cell Count -- methods
- Cells, Cultured
- Cloning, Molecular -- methods
- Cornified Envelope Proline-Rich Proteins
- Desipramine -- pharmacology
- Disease Models, Animal
- Evoked Potentials, Motor -- physiology
- Female
- GPI-Linked Proteins
- Ganglia, Spinal -- cytology
- Glial Fibrillary Acidic Protein -- metabolism
- Glucose -- analogs & derivatives
- Growth Cones -- drug effects
- Immunohistochemistry -- methods
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Motor Activity -- physiology
- Myelin Proteins -- genetics
- Myelin Sheath -- metabolism
- Myelin-Associated Glycoprotein
- Nerve Regeneration
- Neurons -- cytology
- Nogo Proteins
- Nogo Receptor 1
- Phospholipid Ethers -- pharmacology
- Proteins -- metabolism
- Pyramidal Tracts -- physiopathology
- Receptors, Cell Surface -- genetics
- Recovery of Function -- physiology
- Serotonin -- metabolism
- Serotonin Agents -- pharmacology
- Spinal Cord -- metabolism
- Spinal Cord Injuries -- genetics
- Time Factors
No physical items for this record
Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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