Hypertrophy, fibrosis, and sudden cardiac death in response to pathological stimuli in mice with mutations in cardiac troponin T. [electronic resource]
Producer: 20050624Description: 2102-9 p. digitalISSN:- 1524-4539
- Adrenergic alpha-Agonists -- toxicity
- Adrenergic beta-Agonists -- toxicity
- Amino Acid Substitution
- Angiotensin II -- toxicity
- Animals
- Body Weight
- Cardiomyopathy, Hypertrophic, Familial -- complications
- Cell Size
- Death, Sudden, Cardiac -- etiology
- Female
- Fibrosis
- Gene Expression Profiling
- Isoproterenol -- toxicity
- Male
- Mice
- Mice, Inbred C57BL
- Mice, Transgenic
- Mutation
- Mutation, Missense
- Myocytes, Cardiac -- pathology
- Organ Size
- Phenotype
- Phenylephrine -- toxicity
- Point Mutation
- Protein Isoforms -- genetics
- RNA Splice Sites -- genetics
- RNA, Messenger -- biosynthesis
- Sex Characteristics
- Troponin T -- deficiency
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Publication Type: Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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