Heterologous desensitization of insulin secretion by GIP (glucose-dependent insulinotropic peptide) in INS-1 cells: the significance of Galphai2 and investigations on the mechanism involved. [electronic resource]
Producer: 20050804Description: 205-12 p. digitalISSN:- 0263-6484
- 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine -- analogs & derivatives
- 8-Bromo Cyclic Adenosine Monophosphate -- analogs & derivatives
- Adrenergic alpha-Agonists -- pharmacology
- Animals
- Brimonidine Tartrate
- Cell Line, Tumor
- Cyclic AMP-Dependent Protein Kinases -- antagonists & inhibitors
- Enzyme Inhibitors -- pharmacology
- GTP-Binding Protein alpha Subunit, Gi2
- GTP-Binding Protein alpha Subunits, Gi-Go -- genetics
- Gastric Inhibitory Polypeptide -- pharmacology
- Gene Expression -- drug effects
- Glucagon -- metabolism
- Glucagon-Like Peptide 1
- Immunosuppressive Agents -- pharmacology
- Insulin -- metabolism
- Insulin Secretion
- Islets of Langerhans -- cytology
- Peptide Fragments -- metabolism
- Protein Precursors -- metabolism
- Proto-Oncogene Proteins -- genetics
- Quinoxalines -- pharmacology
- RNA, Messenger -- metabolism
- Rats
- Second Messenger Systems -- drug effects
- Tacrolimus -- pharmacology
- Thionucleotides -- pharmacology
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Publication Type: Journal Article
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