Caveolin-1 gene disruption promotes mammary tumorigenesis and dramatically enhances lung metastasis in vivo. Role of Cav-1 in cell invasiveness and matrix metalloproteinase (MMP-2/9) secretion. [electronic resource]
Producer: 20050111Description: 51630-46 p. digitalISSN:- 0021-9258
- Animals
- Caveolin 1
- Caveolins -- genetics
- Cell Line, Tumor
- Cell Membrane -- metabolism
- Cell Movement
- Cells, Cultured
- Collagen -- pharmacology
- Cyclin D1 -- biosynthesis
- Disease Progression
- Drug Combinations
- Electrophoresis, Polyacrylamide Gel
- Enzyme Activation
- Female
- Gene Expression Regulation, Neoplastic
- Immunoblotting
- Inhibitory Concentration 50
- Laminin -- pharmacology
- Lung Neoplasms -- secondary
- Male
- Mammary Neoplasms, Animal -- genetics
- Mammary Tumor Virus, Mouse -- genetics
- Matrix Metalloproteinase 2 -- metabolism
- Matrix Metalloproteinase 9 -- metabolism
- Mice
- Mice, Transgenic
- Microscopy, Fluorescence
- Mitogen-Activated Protein Kinase 1 -- biosynthesis
- Mitogen-Activated Protein Kinase 3 -- biosynthesis
- Mutation
- Neoplasm Invasiveness
- Neoplasm Metastasis -- genetics
- Peptides -- chemistry
- Phosphorylation
- Proteoglycans -- pharmacology
- Recombinant Proteins -- metabolism
- Retinoblastoma Protein -- metabolism
- Retroviridae -- genetics
- Time Factors
- Up-Regulation
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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