Transgenic expression of Bcl-2 modulates energy metabolism, prevents cytosolic acidification during ischemia, and reduces ischemia/reperfusion injury. [electronic resource]
Producer: 20050425Description: 734-41 p. digitalISSN:- 1524-4571
- Adenosine Triphosphate -- metabolism
- Anaerobiosis
- Animals
- Apoptosis
- Blotting, Western
- Cytosol -- chemistry
- Energy Metabolism -- drug effects
- Female
- Gene Expression Regulation
- Genes, bcl-2
- Glycolysis
- Humans
- Hydrogen-Ion Concentration
- Male
- Mice
- Mice, Transgenic
- Mitochondria, Heart -- drug effects
- Myocardial Contraction
- Myocardial Infarction -- pathology
- Myocardial Ischemia -- metabolism
- Myocardial Reperfusion Injury -- metabolism
- Nuclear Magnetic Resonance, Biomolecular
- Oligomycins -- pharmacology
- Phosphocreatine -- metabolism
- Porins -- metabolism
- Proto-Oncogene Proteins c-bcl-2 -- biosynthesis
- Proton-Translocating ATPases -- physiology
- Recombinant Fusion Proteins -- physiology
- Voltage-Dependent Anion Channels
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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