Flagellin acting via TLR5 is the major activator of key signaling pathways leading to NF-kappa B and proinflammatory gene program activation in intestinal epithelial cells. [electronic resource]
Producer: 20050509Description: 33 p. digitalISSN:- 1471-2180
- Adaptor Proteins, Signal Transducing
- Antigens, Differentiation -- metabolism
- Bacterial Proteins -- immunology
- Cell Line, Tumor
- Cells, Cultured
- Central Nervous System Neoplasms -- genetics
- DNA -- metabolism
- Enzyme Activation -- physiology
- Epithelial Cells -- enzymology
- Flagellin -- immunology
- Glioblastoma -- genetics
- HT29 Cells -- chemistry
- HeLa Cells -- chemistry
- Humans
- I-kappa B Kinase
- Inflammation -- genetics
- Intestines -- microbiology
- Kidney -- chemistry
- Lung Neoplasms -- genetics
- Membrane Glycoproteins -- physiology
- Mitogen-Activated Protein Kinase Kinases -- metabolism
- Mitogen-Activated Protein Kinases -- metabolism
- Myeloid Differentiation Factor 88
- NF-kappa B -- metabolism
- Protein Binding
- Protein Serine-Threonine Kinases -- metabolism
- Receptors, Cell Surface -- physiology
- Receptors, Immunologic -- metabolism
- Salmonella -- immunology
- Salmonella Infections -- pathology
- Signal Transduction -- physiology
- Solubility
- Toll-Like Receptor 5
- Toll-Like Receptors
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Publication Type: Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
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