Apc deficiency is associated with increased Egfr activity in the intestinal enterocytes and adenomas of C57BL/6J-Min/+ mice. [electronic resource]
Producer: 20041124Description: 43261-72 p. digitalISSN:- 0021-9258
- Adaptor Proteins, Signal Transducing
- Adenoma -- metabolism
- Adenomatous Polyposis Coli Protein -- deficiency
- Alleles
- Animals
- Anti-Inflammatory Agents, Non-Steroidal -- pharmacology
- Cell Line
- Clathrin -- metabolism
- Colorectal Neoplasms -- metabolism
- Cyclooxygenase 2
- Dinoprostone -- metabolism
- Disease Progression
- Enterocytes -- metabolism
- Enzyme-Linked Immunosorbent Assay
- ErbB Receptors -- metabolism
- Genes, APC
- HeLa Cells
- Humans
- Immunoblotting
- Immunohistochemistry
- Immunoprecipitation
- Intestinal Mucosa -- metabolism
- Intestine, Small -- metabolism
- Isoenzymes -- metabolism
- Jurkat Cells
- Membrane Proteins
- Mice
- Mice, Inbred C57BL
- Mice, Transgenic
- Models, Biological
- Mutation
- Phosphatidylinositol 3-Kinases -- metabolism
- Phosphoproteins -- metabolism
- Phosphorylation
- Prostaglandin-Endoperoxide Synthases -- metabolism
- Protein Serine-Threonine Kinases -- metabolism
- Proto-Oncogene Proteins -- metabolism
- Proto-Oncogene Proteins c-akt
- Recurrence
- Signal Transduction
- Tissue Distribution
- Transcriptional Activation
- Tyrosine -- chemistry
- Ubiquitin -- metabolism
- Up-Regulation
- Vanadates -- pharmacology
- src-Family Kinases -- metabolism
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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