Inhibition of mitochondrial Na+-Ca2+ exchange restores agonist-induced ATP production and Ca2+ handling in human complex I deficiency. [electronic resource]
Producer: 20041026Description: 40328-36 p. digitalISSN:- 0021-9258
- Adenosine Triphosphate -- chemistry
- Bradykinin -- metabolism
- Calcium -- metabolism
- Cell Nucleus -- metabolism
- Clonazepam -- analogs & derivatives
- Cytosol -- metabolism
- Electron Transport Complex I -- deficiency
- Fibroblasts -- metabolism
- Fluorescent Dyes -- pharmacology
- Humans
- Ionomycin -- pharmacology
- Leigh Disease -- metabolism
- Luciferases -- metabolism
- Membrane Potentials
- Microscopy, Fluorescence
- Mitochondria -- metabolism
- Mitochondrial Encephalomyopathies -- metabolism
- Oxygen -- metabolism
- Parkinson Disease -- metabolism
- Skin -- metabolism
- Sodium-Calcium Exchanger -- antagonists & inhibitors
- Thiazepines -- pharmacology
- Time Factors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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