Neutrophil elastase contributes to the development of ischemia-reperfusion-induced liver injury by decreasing endothelial production of prostacyclin in rats. [electronic resource]
Producer: 20041220Description: G1116-23 p. digitalISSN:- 0193-1857
- 6-Ketoprostaglandin F1 alpha -- metabolism
- Animals
- Capillary Permeability -- drug effects
- Cephalosporins -- antagonists & inhibitors
- Chemokines, CXC -- metabolism
- Cyclooxygenase Inhibitors -- pharmacology
- Endothelium -- metabolism
- Enzyme Inhibitors -- pharmacology
- Epoprostenol -- biosynthesis
- Glycine -- analogs & derivatives
- Iloprost -- pharmacology
- Indomethacin -- pharmacology
- Intercellular Signaling Peptides and Proteins -- metabolism
- Leukocyte Elastase -- antagonists & inhibitors
- Liver -- drug effects
- Liver Circulation -- drug effects
- Male
- NG-Nitroarginine Methyl Ester -- pharmacology
- Nitric Oxide -- metabolism
- Nitric Oxide Synthase -- antagonists & inhibitors
- Nitric Oxide Synthase Type III
- Peroxidase -- metabolism
- Rats
- Rats, Wistar
- Reperfusion Injury -- enzymology
- Sulfonamides -- antagonists & inhibitors
- Transaminases -- metabolism
- Vasodilator Agents -- pharmacology
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Publication Type: Journal Article
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