Endoplasmic reticulum stress response is involved in nonsteroidal anti-inflammatory drug-induced apoptosis. [electronic resource]
Producer: 20050214Description: 1009-16 p. digitalISSN:- 1350-9047
- Activating Transcription Factor 4
- Animals
- Anti-Inflammatory Agents, Non-Steroidal -- pharmacology
- Apoptosis
- Blotting, Northern
- Celecoxib
- Cell Line
- Cell Survival
- Cells, Cultured
- DNA-Binding Proteins -- metabolism
- Diclofenac -- pharmacology
- Dose-Response Relationship, Drug
- Endoplasmic Reticulum -- drug effects
- Endoplasmic Reticulum Chaperone BiP
- Enzyme Activation
- Gastric Mucosa -- pathology
- Genes, Reporter
- Guinea Pigs
- Heat-Shock Proteins -- metabolism
- Hydrogen Peroxide -- pharmacology
- Ibuprofen -- pharmacology
- Immunoblotting
- Indomethacin -- pharmacology
- Luciferases -- metabolism
- Macrophages -- metabolism
- Microscopy, Fluorescence
- Molecular Chaperones -- metabolism
- Plasmids -- metabolism
- Pyrazoles -- pharmacology
- RNA, Messenger -- metabolism
- Regulatory Factor X Transcription Factors
- Sulfonamides -- pharmacology
- Time Factors
- Transcription Factors -- metabolism
- Transcription, Genetic
- Transfection
- X-Box Binding Protein 1
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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