Rescue of TNFalpha-inhibited neuronal cells by IGF-1 involves Akt and c-Jun N-terminal kinases. [electronic resource]
Producer: 20040621Description: 466-74 p. digitalISSN:- 0360-4012
- 3-Phosphoinositide-Dependent Protein Kinases
- Animals
- Antioxidants -- pharmacology
- Butylated Hydroxyanisole -- pharmacology
- Caspases -- metabolism
- Cell Death -- drug effects
- Cell Division -- drug effects
- Cell Survival -- drug effects
- Chromones -- pharmacology
- Dicumarol -- pharmacology
- Dose-Response Relationship, Drug
- Drug Interactions
- Enzyme Inhibitors -- pharmacology
- Forkhead Box Protein O1
- Forkhead Transcription Factors
- Humans
- Insulin-Like Growth Factor I -- pharmacology
- JNK Mitogen-Activated Protein Kinases
- Mitogen-Activated Protein Kinases -- physiology
- Morpholines -- pharmacology
- Neuroblastoma
- Neurons -- drug effects
- Oligopeptides -- metabolism
- PC12 Cells
- Phosphorylation -- drug effects
- Poly (ADP-Ribose) Polymerase-1
- Poly(ADP-ribose) Polymerases
- Protein Serine-Threonine Kinases -- metabolism
- Proteins -- metabolism
- Proto-Oncogene Proteins -- physiology
- Proto-Oncogene Proteins c-akt
- Rats
- Time Factors
- Transcription Factors -- metabolism
- Tumor Necrosis Factor-alpha -- toxicity
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Publication Type: Comparative Study; Journal Article
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