G-CSF receptor truncations found in SCN/AML relieve SOCS3-controlled inhibition of STAT5 but leave suppression of STAT3 intact. [electronic resource]
Producer: 20040830Description: 667-74 p. digitalISSN:- 0006-4971
- Amino Acid Sequence
- Animals
- Binding Sites
- DNA-Binding Proteins -- antagonists & inhibitors
- Disease Models, Animal
- Humans
- Leukemia, Myeloid, Acute -- genetics
- Mice
- Mice, Mutant Strains
- Milk Proteins
- Neutropenia -- genetics
- Receptors, Granulocyte Colony-Stimulating Factor -- genetics
- Repressor Proteins -- genetics
- STAT3 Transcription Factor
- STAT5 Transcription Factor
- Sequence Deletion
- Signal Transduction
- Suppressor of Cytokine Signaling 3 Protein
- Suppressor of Cytokine Signaling Proteins
- Trans-Activators -- antagonists & inhibitors
- Transcription Factors -- genetics
- Tyrosine
- src Homology Domains
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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