The extracellular matrix, p53 and estrogen compete to regulate cell-surface Fas/Apo-1 suicide receptor expression in proliferating embryonic cerebral cortical precursors, and reciprocally, Fas-ligand modifies estrogen control of cell-cycle proteins. [electronic resource]
Producer: 20040506Description: 11 p. digitalISSN:- 1471-2202
- Animals
- Anoikis -- physiology
- Apoptosis -- physiology
- Blotting, Western
- CASP8 and FADD-Like Apoptosis Regulating Protein
- Carrier Proteins -- biosynthesis
- Cell Cycle -- physiology
- Cell Cycle Proteins -- metabolism
- Cell Nucleus -- metabolism
- Cell-Matrix Junctions -- metabolism
- Cells, Cultured
- Cerebral Cortex -- cytology
- Cyclin-Dependent Kinase Inhibitor p21
- Cyclins -- metabolism
- Estrogens -- pharmacology
- Extracellular Matrix -- metabolism
- Fas Ligand Protein
- Flow Cytometry
- Intracellular Signaling Peptides and Proteins
- Membrane Glycoproteins -- physiology
- Neurons -- classification
- Proliferating Cell Nuclear Antigen -- metabolism
- Rats
- Rats, Sprague-Dawley
- Stem Cells -- cytology
- Tumor Suppressor Protein p53 -- metabolism
- fas Receptor -- metabolism
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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