Direct inhibition of the mitochondrial permeability transition pore: a possible mechanism responsible for anti-apoptotic effects of melatonin. [electronic resource]
Producer: 20040928Description: 869-71 p. digitalISSN:- 1530-6860
- Animals
- Apoptosis -- drug effects
- Calcium Signaling -- drug effects
- Cell Hypoxia
- Corpus Striatum -- cytology
- Infarction, Middle Cerebral Artery -- drug therapy
- Inhibitory Concentration 50
- Ion Channels -- antagonists & inhibitors
- Ischemic Attack, Transient -- drug therapy
- Male
- Melatonin -- pharmacology
- Mice
- Mitochondria, Liver -- drug effects
- Mitochondrial Membrane Transport Proteins
- Mitochondrial Permeability Transition Pore
- Models, Animal
- N-Methylaspartate -- toxicity
- Neuroprotective Agents -- pharmacology
- Patch-Clamp Techniques
- Rats
- Rats, Sprague-Dawley
- Rats, Wistar
- Receptors, N-Methyl-D-Aspartate -- drug effects
- Reperfusion Injury -- drug therapy
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Publication Type: Journal Article
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