Simvastatin prevents load-induced protein tyrosine nitration in overloaded hearts. [electronic resource]
Producer: 20041008Description: 1060-6 p. digitalISSN:- 1524-4563
- Animals
- Antioxidants -- pharmacology
- Aorta
- Constriction
- Coronary Vessels -- enzymology
- Enzyme Induction
- Hydroxymethylglutaryl-CoA Reductase Inhibitors -- pharmacology
- Hypertrophy, Left Ventricular -- drug therapy
- Isoenzymes -- metabolism
- Male
- Membrane Transport Proteins -- metabolism
- Mitogen-Activated Protein Kinase 1 -- metabolism
- Mitogen-Activated Protein Kinase 3
- Mitogen-Activated Protein Kinases -- metabolism
- NADPH Dehydrogenase -- metabolism
- NADPH Oxidases
- Nitric Oxide Synthase -- metabolism
- Oxidative Stress
- Phosphoproteins -- metabolism
- Pressure
- Proteins -- metabolism
- Rats
- Rats, Wistar
- Simvastatin -- pharmacology
- Tyrosine -- analogs & derivatives
- Ventricular Remodeling -- physiology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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