Growth factor deprivation induces an alternative non-apoptotic death mechanism that is inhibited by Bcl2 in cells derived from neural precursor cells. [electronic resource]
Producer: 20040914Description: 735-48 p. digitalISSN:- 1525-8165
- Adenine -- analogs & derivatives
- Amino Acid Chloromethyl Ketones -- pharmacology
- Animals
- Apoptosis -- drug effects
- Apoptosis Regulatory Proteins
- Autophagy -- drug effects
- Beclin-1
- Caspase Inhibitors
- Cell Death -- drug effects
- Cell Differentiation -- drug effects
- Cell Survival -- drug effects
- Chromones -- pharmacology
- Cycloheximide -- pharmacology
- Enzyme Inhibitors -- pharmacology
- Epidermal Growth Factor -- pharmacology
- Fibroblast Growth Factor 2 -- deficiency
- Gene Expression Regulation -- genetics
- Glial Fibrillary Acidic Protein -- analysis
- Green Fluorescent Proteins
- Immunohistochemistry
- Intermediate Filament Proteins -- analysis
- Luminescent Proteins -- genetics
- Mesencephalon -- cytology
- Mice
- Microscopy, Electron
- Morpholines -- pharmacology
- Nerve Tissue Proteins -- analysis
- Nestin
- Neurons -- cytology
- Proteins -- genetics
- Proto-Oncogene Proteins -- genetics
- Proto-Oncogene Proteins c-bcl-2
- Stem Cells -- cytology
- Transformation, Genetic -- genetics
- Tubulin -- analysis
- Vacuoles -- drug effects
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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