Fluvastatin prevents nephropathy likely through suppression of connective tissue growth factor-mediated extracellular matrix accumulation. [electronic resource]
Producer: 20040224Description: 66-75 p. digitalISSN:- 0014-4800
- Animals
- Anticholesteremic Agents -- therapeutic use
- Blood Urea Nitrogen
- Connective Tissue Growth Factor
- Creatinine -- urine
- Disease Models, Animal
- Extracellular Matrix -- metabolism
- Fatty Acids, Monounsaturated -- therapeutic use
- Fluvastatin
- Immediate-Early Proteins -- biosynthesis
- Indoles -- therapeutic use
- Intercellular Signaling Peptides and Proteins -- biosynthesis
- Kidney Failure, Chronic -- metabolism
- Kidney Glomerulus -- drug effects
- Male
- Matrix Metalloproteinase Inhibitors
- Nephrectomy
- Proteinuria
- RNA, Messenger -- metabolism
- Rats
- Rats, Wistar
- Tissue Inhibitor of Metalloproteinase-2 -- biosynthesis
- Up-Regulation
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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