Axonal defects in mouse models of motoneuron disease. [electronic resource]
Producer: 20040322Description: 272-86 p. digitalISSN:- 0022-3034
- Aged
- Alternative Splicing -- physiology
- Amyotrophic Lateral Sclerosis -- genetics
- Animals
- Axonal Transport
- Axons -- metabolism
- Carrier Proteins -- metabolism
- Caspase 2
- Caspases -- genetics
- Cell Survival
- Cells, Cultured
- Ciliary Neurotrophic Factor -- deficiency
- Cyclic AMP Response Element-Binding Protein
- DNA-Binding Proteins
- Disease Models, Animal
- Embryo, Mammalian
- Fungal Proteins -- genetics
- Genes, rRNA -- physiology
- HSP70 Heat-Shock Proteins -- metabolism
- Heterogeneous-Nuclear Ribonucleoproteins -- metabolism
- Humans
- Immunohistochemistry
- Mice
- Mice, Transgenic
- Middle Aged
- Motor Neuron Disease -- genetics
- Mutation
- Nerve Growth Factor -- metabolism
- Nerve Tissue Proteins -- metabolism
- Precipitin Tests
- Protein Serine-Threonine Kinases
- Proto-Oncogene Proteins -- metabolism
- Proto-Oncogene Proteins A-raf
- Proto-Oncogene Proteins c-akt
- Proto-Oncogene Proteins c-raf -- metabolism
- RNA, Messenger -- biosynthesis
- RNA-Binding Proteins
- Reverse Transcriptase Polymerase Chain Reaction -- methods
- SMN Complex Proteins
- Sciatic Nerve -- metabolism
- Spine -- metabolism
- Superoxide Dismutase -- genetics
- Superoxide Dismutase-1
- Transcription Factors -- genetics
- Tubulin -- metabolism
- tau Proteins -- metabolism
No physical items for this record
Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
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