The susceptibility to experimental myasthenia gravis of STAT6-/- and STAT4-/- BALB/c mice suggests a pathogenic role of Th1 cells. [electronic resource]
Producer: 20040402Description: 97-103 p. digitalISSN:- 0022-1767
- Animals
- Autoantibodies -- biosynthesis
- CD4-Positive T-Lymphocytes -- cytology
- Cell Division -- immunology
- DNA-Binding Proteins -- deficiency
- Female
- Genetic Predisposition to Disease
- Immunoglobulin G -- biosynthesis
- Interferon-gamma -- metabolism
- Interleukin-4 -- metabolism
- Mice
- Mice, Inbred BALB C
- Mice, Knockout
- Myasthenia Gravis, Autoimmune, Experimental -- genetics
- Peptide Fragments -- pharmacology
- Protein Subunits -- pharmacology
- Receptors, Nicotinic -- immunology
- STAT4 Transcription Factor
- STAT6 Transcription Factor
- Signal Transduction -- genetics
- Th1 Cells -- immunology
- Torpedo -- immunology
- Trans-Activators -- deficiency
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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