Hypoxia increases Hsp90 binding to eNOS via PI3K-Akt in porcine coronary artery endothelium. [electronic resource]
Producer: 20040324Description: 182-90 p. digitalISSN:- 0023-6837
- Androstadienes -- pharmacology
- Animals
- Benzoquinones
- Cell Hypoxia -- physiology
- Cells, Cultured
- Coronary Vessels -- enzymology
- Endothelium, Vascular -- enzymology
- Enzyme Inhibitors -- pharmacology
- Flavonoids -- pharmacology
- HSP90 Heat-Shock Proteins -- antagonists & inhibitors
- Lactams, Macrocyclic
- Mitogen-Activated Protein Kinase 1 -- antagonists & inhibitors
- Mitogen-Activated Protein Kinase 3
- Mitogen-Activated Protein Kinases -- antagonists & inhibitors
- Nitric Oxide -- metabolism
- Nitric Oxide Synthase -- metabolism
- Nitric Oxide Synthase Type III
- Phosphatidylinositol 3-Kinases -- metabolism
- Phosphorylation
- Protein Serine-Threonine Kinases
- Proto-Oncogene Proteins -- metabolism
- Proto-Oncogene Proteins c-akt
- Quinones -- pharmacology
- Swine
- Time Factors
- Up-Regulation
- Wortmannin
No physical items for this record
Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
There are no comments on this title.
Log in to your account to post a comment.