Contribution of Kir3.1, Kir3.2A and Kir3.2C subunits to native G protein-gated inwardly rectifying potassium currents in cultured hippocampal neurons. [electronic resource]
Producer: 20031223Description: 2110-8 p. digitalISSN:- 0953-816X
- Adenosine -- pharmacology
- Analgesics -- pharmacology
- Animals
- Animals, Newborn
- Baclofen -- pharmacology
- Benzoxazines
- Blotting, Northern
- Calcium Channel Blockers -- pharmacology
- Carbachol -- pharmacology
- Cell Cycle Proteins -- metabolism
- Cells, Cultured
- Cholinergic Agonists -- pharmacology
- Dose-Response Relationship, Drug
- Drug Interactions
- Embryo, Mammalian
- Free Radical Scavengers -- pharmacology
- G Protein-Coupled Inwardly-Rectifying Potassium Channels
- GABA Agonists -- pharmacology
- GTP-Binding Proteins -- physiology
- Hippocampus -- cytology
- Hormones -- pharmacology
- Humans
- Kidney
- Membrane Potentials -- drug effects
- Microscopy, Confocal -- instrumentation
- Morpholines -- pharmacology
- Naphthalenes -- pharmacology
- Neurons -- drug effects
- Patch-Clamp Techniques -- methods
- Pertussis Toxin -- pharmacology
- Potassium Channels -- metabolism
- Potassium Channels, Inwardly Rectifying -- classification
- Protein Subunits -- physiology
- RNA, Messenger -- biosynthesis
- Rats
- Rats, Sprague-Dawley
- Receptors, G-Protein-Coupled -- metabolism
- Reverse Transcriptase Polymerase Chain Reaction -- methods
- Serotonin -- pharmacology
- Somatostatin -- pharmacology
- Time Factors
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Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
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