Proteasomal degradation of Runx2 shortens parathyroid hormone-induced anti-apoptotic signaling in osteoblasts. A putative explanation for why intermittent administration is needed for bone anabolism. [electronic resource]
Producer: 20040115Description: 50259-72 p. digitalISSN:- 0021-9258
- Animals
- Apoptosis
- Blotting, Western
- Bone Resorption
- Calcium -- metabolism
- Carrier Proteins -- metabolism
- Cells, Cultured
- Core Binding Factor Alpha 1 Subunit
- Culture Media, Conditioned -- pharmacology
- Cyclic AMP Response Element-Binding Protein -- metabolism
- Cyclic AMP-Dependent Protein Kinases -- metabolism
- Cysteine Endopeptidases -- metabolism
- Dactinomycin -- pharmacology
- Female
- HeLa Cells
- Humans
- Kinetics
- Membrane Glycoproteins -- metabolism
- Mice
- Mice, Inbred C57BL
- Models, Biological
- Models, Genetic
- Multienzyme Complexes -- metabolism
- Neoplasm Proteins
- Osteoblasts -- metabolism
- Parathyroid Hormone -- metabolism
- Phosphorylation
- Proteasome Endopeptidase Complex
- Proto-Oncogene Proteins c-bcl-2 -- metabolism
- RANK Ligand
- RNA -- metabolism
- RNA, Messenger -- metabolism
- Receptor Activator of Nuclear Factor-kappa B
- Signal Transduction
- Time Factors
- Transcription Factors -- metabolism
- Transcription, Genetic
- bcl-Associated Death Protein
No physical items for this record
Publication Type: Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
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