Increased intracellular cyclic AMP inhibits inositol phospholipid hydrolysis induced by perturbation of the T cell receptor/CD3 complex but not by G-protein stimulation. Association with protein kinase A-mediated phosphorylation of phospholipase C-gamma 1. [electronic resource]
Producer: 19920707Description: 189-99 p. digitalISSN:- 0264-6021
- Animals
- Antigens, Differentiation, T-Lymphocyte -- drug effects
- CD3 Complex
- Cattle
- Cell Membrane Permeability -- drug effects
- Cholera Toxin -- pharmacology
- Colforsin -- analogs & derivatives
- Cricetinae
- Cyclic AMP -- biosynthesis
- Dinoprostone -- pharmacology
- Diterpenes
- Down-Regulation -- physiology
- Enzyme Activation
- GTP-Binding Proteins -- drug effects
- Hydrolysis
- Inositol Phosphates -- metabolism
- Intracellular Fluid -- metabolism
- Isoenzymes -- metabolism
- Mice
- Mice, Nude
- Phospholipids -- metabolism
- Phosphorylation
- Protein Kinases -- metabolism
- Receptors, Antigen, T-Cell -- antagonists & inhibitors
- Stimulation, Chemical
- T-Lymphocytes -- drug effects
- T-Lymphocytes, Helper-Inducer -- drug effects
- Type C Phospholipases -- metabolism
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Publication Type: Journal Article
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