A failure to repair self-proteins leads to T cell hyperproliferation and autoantibody production. [electronic resource]
Producer: 20031204Description: 2840-7 p. digitalISSN:- 0022-1767
- Animals
- Autoantibodies -- biosynthesis
- Autoantigens -- immunology
- Bone Marrow Transplantation -- immunology
- CD28 Antigens -- pharmacology
- Cell Division -- genetics
- Immunophenotyping
- Isoaspartic Acid -- metabolism
- Lupus Erythematosus, Systemic -- genetics
- Lymph Nodes -- enzymology
- Lymphocyte Activation -- genetics
- Lymphoid Tissue -- enzymology
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Mitogens -- pharmacology
- Phosphorylation
- Protein D-Aspartate-L-Isoaspartate Methyltransferase -- deficiency
- Receptors, Antigen, T-Cell -- physiology
- Signal Transduction -- genetics
- T-Lymphocytes -- enzymology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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