BCL6 overexpression prevents increase in reactive oxygen species and inhibits apoptosis induced by chemotherapeutic reagents in B-cell lymphoma cells. [electronic resource]
Producer: 20030819Description: 4459-68 p. digitalISSN:- 0950-9232
- Acetylcysteine -- pharmacology
- Antineoplastic Agents -- pharmacology
- Antioxidants -- pharmacology
- Apoptosis -- drug effects
- Cysteine Endopeptidases -- drug effects
- Cysteine Proteinase Inhibitors -- pharmacology
- DNA-Binding Proteins -- drug effects
- Doxycycline -- pharmacology
- Etoposide -- pharmacology
- Free Radical Scavengers -- pharmacology
- Gene Expression Regulation, Neoplastic -- drug effects
- Humans
- Leupeptins -- pharmacology
- Lymphoma, B-Cell -- drug therapy
- Membrane Potentials -- drug effects
- Mitochondria -- drug effects
- Multienzyme Complexes -- antagonists & inhibitors
- Mutation
- Proteasome Endopeptidase Complex
- Proto-Oncogene Proteins -- drug effects
- Proto-Oncogene Proteins c-bcl-6
- Reactive Oxygen Species -- metabolism
- Transcription Factors -- drug effects
- Tumor Cells, Cultured
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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