Oxidative stress induces nuclear loss of DNA repair proteins Ku70 and Ku80 and apoptosis in pancreatic acinar AR42J cells. [electronic resource]
Producer: 20031120Description: 36676-87 p. digitalISSN:- 0021-9258
- Antigens, Nuclear -- biosynthesis
- Apoptosis
- Blotting, Western
- Caspase 3
- Caspase Inhibitors
- Cell Death
- Cell Line, Tumor
- Cell Nucleus -- metabolism
- Cell Survival
- Cytoplasm -- metabolism
- DNA Fragmentation
- DNA Helicases
- DNA Repair
- DNA, Complementary -- metabolism
- DNA-Binding Proteins -- biosynthesis
- Dose-Response Relationship, Drug
- Enzyme Inhibitors -- pharmacology
- Genes, Dominant
- Glucose Oxidase -- metabolism
- Glutathione Transferase -- metabolism
- Humans
- Hydrogen Peroxide -- pharmacology
- Ku Autoantigen
- Microscopy, Fluorescence
- Oxidative Stress
- Pancreas -- cytology
- Plasmids -- metabolism
- Protein Binding
- Proto-Oncogene Proteins -- metabolism
- Proto-Oncogene Proteins c-bcl-2 -- metabolism
- Recombinant Fusion Proteins -- metabolism
- Time Factors
- Transfection
- Tumor Suppressor Protein p53 -- metabolism
- alpha Karyopherins -- chemistry
- bcl-2-Associated X Protein
- beta Karyopherins -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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