Hepatitis B virus X protein enhances transcriptional activity of hypoxia-inducible factor-1alpha through activation of mitogen-activated protein kinase pathway. [electronic resource]
Producer: 20031106Description: 39076-84 p. digitalISSN:- 0021-9258
- Active Transport, Cell Nucleus
- Androstadienes -- pharmacology
- Animals
- Blotting, Western
- Cell Line
- Cell Nucleus -- metabolism
- Cobalt -- pharmacology
- Endothelial Growth Factors -- metabolism
- Enzyme Inhibitors -- pharmacology
- Flavonoids -- pharmacology
- Flow Cytometry
- Genes, Reporter
- HeLa Cells
- Humans
- Hypoxia
- Hypoxia-Inducible Factor 1, alpha Subunit
- Immunohistochemistry
- Intercellular Signaling Peptides and Proteins -- metabolism
- Ligases -- metabolism
- Lymphokines -- metabolism
- MAP Kinase Signaling System
- Mice
- Mice, Transgenic
- Microscopy, Fluorescence
- Mitogen-Activated Protein Kinase 1 -- metabolism
- Mitogen-Activated Protein Kinase 3
- Mitogen-Activated Protein Kinases -- metabolism
- Phosphorylation
- Plasmids -- metabolism
- Precipitin Tests
- Promoter Regions, Genetic
- Protein Transport
- Reverse Transcriptase Polymerase Chain Reaction
- Signal Transduction
- Time Factors
- Trans-Activators -- metabolism
- Transcription Factors -- metabolism
- Transcription, Genetic
- Transcriptional Activation
- Transfection
- Tumor Cells, Cultured
- Tumor Suppressor Proteins
- Ubiquitin-Protein Ligases
- Vascular Endothelial Growth Factor A
- Vascular Endothelial Growth Factors
- Viral Regulatory and Accessory Proteins
- Von Hippel-Lindau Tumor Suppressor Protein
- Wortmannin
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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