Dieldrin induces apoptosis by promoting caspase-3-dependent proteolytic cleavage of protein kinase Cdelta in dopaminergic cells: relevance to oxidative stress and dopaminergic degeneration. [electronic resource]
Producer: 20030905Description: 945-64 p. digitalISSN:- 0306-4522
- Animals
- Apoptosis -- drug effects
- Caspase 3
- Caspases -- drug effects
- Cytochrome c Group -- metabolism
- Dieldrin -- pharmacology
- Dopamine -- metabolism
- Dose-Response Relationship, Drug
- Enzyme Inhibitors -- pharmacology
- Feedback, Physiological -- drug effects
- Free Radical Scavengers -- pharmacology
- Gene Expression Regulation, Enzymologic -- drug effects
- Metalloporphyrins -- pharmacology
- Neurons -- drug effects
- Oxidative Stress -- drug effects
- PC12 Cells
- Parkinsonian Disorders -- enzymology
- Peptide Hydrolases -- drug effects
- Protein Kinase C -- genetics
- Protein Kinase C-delta
- Protein Subunits -- pharmacology
- Rats
- Reaction Time -- drug effects
- Superoxide Dismutase -- metabolism
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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