Consequences of elevated luteinizing hormone on diverse physiological systems: use of the LHbetaCTP transgenic mouse as a model of ovarian hyperstimulation-induced pathophysiology. [electronic resource]
Producer: 20030731Description: 343-75 p. digitalISSN:- 0079-9963
- Aging
- Animals
- Chorionic Gonadotropin, beta Subunit, Human -- genetics
- Cushing Syndrome -- etiology
- Disease Models, Animal
- Female
- Luteinizing Hormone -- genetics
- Luteinizing Hormone, beta Subunit -- genetics
- Mice
- Mice, Transgenic
- Ovarian Diseases -- etiology
- Ovarian Neoplasms -- etiology
- Ovary -- physiopathology
- Peptide Fragments -- genetics
- Polycystic Ovary Syndrome -- etiology
- Primary Ovarian Insufficiency -- etiology
- Recombinant Fusion Proteins
No physical items for this record
Publication Type: Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.; Review
There are no comments on this title.
Log in to your account to post a comment.