Deletion of N-terminal residues 23-88 from prion protein (PrP) abrogates the potential to rescue PrP-deficient mice from PrP-like protein/doppel-induced Neurodegeneration. [electronic resource]
Producer: 20030910Description: 28944-9 p. digitalISSN:- 0021-9258
- Animals
- Ataxia -- genetics
- Brain -- enzymology
- Calbindins
- Cell Death -- genetics
- Cricetinae
- Detergents
- GPI-Linked Proteins
- Gene Deletion
- Gene Expression
- Glutathione Peroxidase -- metabolism
- Mice
- Mice, Transgenic
- Nerve Degeneration -- chemically induced
- Neurons -- metabolism
- Peptide Fragments -- chemistry
- Phenotype
- Pregnancy Proteins -- chemistry
- Prions -- genetics
- Purkinje Cells -- pathology
- Repetitive Sequences, Nucleic Acid
- S100 Calcium Binding Protein G -- analysis
- Solubility
- Superoxide Dismutase -- metabolism
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.