Fractalkine (CX3CL1) stimulated by nuclear factor kappaB (NF-kappaB)-dependent inflammatory signals induces aortic smooth muscle cell proliferation through an autocrine pathway. [electronic resource]
Producer: 20030815Description: 547-58 p. digitalISSN:- 0264-6021
- Androstadienes -- pharmacology
- Animals
- Antineoplastic Agents -- pharmacology
- Aorta -- cytology
- Autocrine Communication
- CX3C Chemokine Receptor 1
- Cell Division -- drug effects
- Cells, Cultured
- Chemokine CX3CL1
- Chemokines, CX3C -- metabolism
- Electrophoretic Mobility Shift Assay
- Enzyme Inhibitors -- pharmacology
- GTP-Binding Proteins -- metabolism
- I-kappa B Proteins -- metabolism
- Luciferases -- metabolism
- Membrane Proteins -- metabolism
- Muscle, Smooth, Vascular -- cytology
- NF-kappa B -- metabolism
- Phosphatidylinositol 3-Kinases -- metabolism
- Phosphoinositide-3 Kinase Inhibitors
- Proline -- analogs & derivatives
- Protein Serine-Threonine Kinases
- Proteins -- metabolism
- Proto-Oncogene Proteins -- antagonists & inhibitors
- Proto-Oncogene Proteins c-akt
- RNA, Messenger -- metabolism
- Rats
- Receptors, Cytokine -- metabolism
- Receptors, HIV -- metabolism
- TNF Receptor-Associated Factor 2
- TNF Receptor-Associated Factor 6
- Thiocarbamates -- pharmacology
- Transfection
- Tumor Necrosis Factor-alpha -- pharmacology
- Wortmannin
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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