Loss of p16INK4a results in increased glucocorticoid receptor activity during fibrosarcoma development. [electronic resource]
Producer: 20030421Description: 3113-8 p. digitalISSN:- 0027-8424
- Animals
- Base Sequence
- Cell Division -- drug effects
- Cyclin-Dependent Kinase Inhibitor p16 -- deficiency
- DNA Methylation
- DNA, Neoplasm -- chemistry
- Dexamethasone -- pharmacology
- Fibrosarcoma -- etiology
- Genes, p16
- Mice
- Precancerous Conditions -- etiology
- Receptors, Glucocorticoid -- genetics
- Transcriptional Activation
- Tumor Cells, Cultured
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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