Lymphotoxin and lipopolysaccharide induce NF-kappaB-p52 generation by a co-translational mechanism. [electronic resource]
Producer: 20030714Description: 82-7 p. digitalISSN:- 1469-221X
- Adaptor Proteins, Signal Transducing
- Adenocarcinoma -- pathology
- B-Lymphocytes -- drug effects
- Breast Neoplasms -- pathology
- DNA -- metabolism
- Dendritic Cells -- drug effects
- Enzyme Activation
- Female
- HeLa Cells -- drug effects
- Humans
- I-kappa B Proteins -- chemistry
- Interleukin-1 -- pharmacology
- Lipopolysaccharides -- pharmacology
- Lymphotoxin beta Receptor
- Lymphotoxin-alpha -- agonists
- Lymphotoxin-beta
- Membrane Proteins -- agonists
- NF-KappaB Inhibitor alpha
- NF-kappa B -- metabolism
- NF-kappa B p52 Subunit
- Protein Binding -- drug effects
- Protein Precursors -- metabolism
- Protein Processing, Post-Translational
- Receptors, Tumor Necrosis Factor -- agonists
- Recombinant Fusion Proteins -- genetics
- Ribosomes -- metabolism
- Sequence Deletion
- Tetradecanoylphorbol Acetate -- pharmacology
- Trans-Activators -- metabolism
- Transcription Factors
- Tumor Cells, Cultured -- drug effects
- Tumor Necrosis Factor Ligand Superfamily Member 14
- Tumor Necrosis Factor-alpha -- pharmacology
- Ubiquitin -- metabolism
No physical items for this record
Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.