Growth differentiation factor-15 prevents low potassium-induced cell death of cerebellar granule neurons by differential regulation of Akt and ERK pathways. [electronic resource]
Producer: 20030514Description: 8904-12 p. digitalISSN:- 0021-9258
- Androstadienes -- pharmacology
- Animals
- Apoptosis
- Blotting, Western
- Brain -- cytology
- Butadienes -- pharmacology
- Cell Death
- Cell Differentiation
- Cell Division
- Cell Survival
- Cells, Cultured
- Chromones -- pharmacology
- Cytokines -- physiology
- DNA, Complementary -- metabolism
- Dose-Response Relationship, Drug
- Enzyme Inhibitors -- pharmacology
- Growth Differentiation Factor 15
- Humans
- Immunoblotting
- In Situ Nick-End Labeling
- JNK Mitogen-Activated Protein Kinases
- L-Lactate Dehydrogenase -- metabolism
- MAP Kinase Signaling System
- Mitogen-Activated Protein Kinases -- metabolism
- Morpholines -- pharmacology
- Neurons -- metabolism
- Nitriles -- pharmacology
- Oxygen -- metabolism
- Phosphatidylinositol 3-Kinases -- metabolism
- Phosphorylation
- Potassium -- metabolism
- Propidium -- pharmacology
- Protein Serine-Threonine Kinases
- Proto-Oncogene Proteins -- metabolism
- Proto-Oncogene Proteins c-akt
- Rats
- Rats, Wistar
- Reactive Oxygen Species
- Recombinant Proteins -- metabolism
- Time Factors
- Wortmannin
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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