Induction of in vitro reprogramming by Toll-like receptor (TLR)2 and TLR4 agonists in murine macrophages: effects of TLR "homotolerance" versus "heterotolerance" on NF-kappa B signaling pathway components. [electronic resource]
Producer: 20030305Description: 508-19 p. digitalISSN:- 0022-1767
- Animals
- Cell Line
- Cells, Cultured
- DNA-Binding Proteins -- antagonists & inhibitors
- Down-Regulation -- genetics
- Drosophila Proteins
- Enzyme Activation -- immunology
- Female
- Host Cell Factor C1
- Humans
- I-kappa B Kinase
- Immune Tolerance -- genetics
- Interferon-beta -- antagonists & inhibitors
- Interleukin-1 Receptor-Associated Kinases
- JNK Mitogen-Activated Protein Kinases
- Lipopolysaccharides -- pharmacology
- Macrophages, Peritoneal -- enzymology
- Membrane Glycoproteins -- agonists
- Mice
- Mice, Inbred C3H
- Mice, Inbred C57BL
- Mitogen-Activated Protein Kinases -- metabolism
- NF-kappa B -- antagonists & inhibitors
- NF-kappa B p50 Subunit
- Octamer Transcription Factor-1
- Protein Kinase Inhibitors
- Protein Kinases -- metabolism
- Protein Serine-Threonine Kinases -- antagonists & inhibitors
- RNA, Messenger -- antagonists & inhibitors
- Receptors, Cell Surface -- agonists
- Sequence Deletion
- Signal Transduction -- genetics
- Toll-Like Receptor 2
- Toll-Like Receptor 4
- Toll-Like Receptors
- Trans-Activators -- antagonists & inhibitors
- Transcription Factor AP-1 -- metabolism
- Transcription Factor RelA
- Transcription Factors -- metabolism
- Tumor Necrosis Factor-alpha -- antagonists & inhibitors
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Publication Type: Comparative Study; Journal Article; Research Support, U.S. Gov't, P.H.S.
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