The stability of the G protein-coupled receptor-beta-arrestin interaction determines the mechanism and functional consequence of ERK activation. [electronic resource]
Producer: 20030422Description: 6258-67 p. digitalISSN:- 0021-9258
- Animals
- Arrestins -- genetics
- COS Cells
- Chlorocebus aethiops
- Epidermal Growth Factor -- pharmacology
- GTP-Binding Proteins -- metabolism
- Green Fluorescent Proteins
- Kinetics
- Luminescent Proteins -- genetics
- MAP Kinase Signaling System -- drug effects
- Mitogen-Activated Protein Kinases -- metabolism
- Models, Biological
- Phosphorylation
- Receptors, Cell Surface -- genetics
- Recombinant Fusion Proteins -- metabolism
- Recombinant Proteins -- metabolism
- Signal Transduction -- physiology
- Transfection
- Vasopressins -- pharmacology
- beta-Arrestins
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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