Suppression of myeloid transcription factors and induction of STAT response genes by AML-specific Flt3 mutations. [electronic resource]
Producer: 20030618Description: 3164-73 p. digitalISSN:- 0006-4971
- Acute Disease
- Animals
- CCAAT-Enhancer-Binding Protein-alpha -- biosynthesis
- Cell Line -- metabolism
- Cluster Analysis
- Computer Systems
- Gene Expression Profiling
- Gene Expression Regulation
- Gene Expression Regulation, Leukemic
- Genes, Synthetic
- Hematopoietic Stem Cells -- metabolism
- Humans
- Interleukin-3 -- pharmacology
- Leukemia, Myeloid -- genetics
- Mice
- Myeloid Cells -- metabolism
- Neoplasm Proteins -- biosynthesis
- Oligonucleotide Array Sequence Analysis
- Protein Isoforms -- physiology
- Protein Serine-Threonine Kinases
- Proto-Oncogene Proteins -- biosynthesis
- Receptor Protein-Tyrosine Kinases -- genetics
- Recombinant Fusion Proteins -- genetics
- Reverse Transcriptase Polymerase Chain Reaction
- Tandem Repeat Sequences
- Trans-Activators -- biosynthesis
- Transcription, Genetic
- Transfection
- fms-Like Tyrosine Kinase 3
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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